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Ipratropium bromide

Catalog #: 0692
3 Citations Datasheet / COA / SDS

Discontinued Product

0692 has been discontinued.
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Ipratropium bromide | CAS No. 22254-24-6 | Non-selective mAChR Antagonists
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Description: Muscarinic antagonist

Chemical Name: (endo,syn)-(±)-3-(3-Hydroxy-1-oxo-2-phenylpropoxy)-8-methyl-8-(1-methylethyl)-8-azoniabicyclo[3.2.1]octane bromide

Purity: ≥98%

Product Details
Citations (3)
Reviews
Biological Activity Technical Data Background Product Datasheets Calculators

Biological Activity

Ipratropium bromide is a muscarinic antagonist, bronchodilator. N-Isopropyl salt of atropine.

Technical Data

M.Wt:
412.37
Formula:
C20H30BrNO3
Solubility:
Soluble to 50 mM in water
Purity:
≥98%
Storage:
Store at +4°C
CAS No:
22254-24-6

The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.
Tocris products are intended for laboratory research use only, unless stated otherwise.

Background References

  1. High-throughput genotoxicity assay identifies antioxidants as inducers of DNA damage response and cell death.
    Fox JT, Sakamuru S, Huang R
    Proc. Natl. Acad. Sci. U.S.A., 2012;109(14):5423-8.
  2. Ipratropium bromide potentiates bronchoconstriction induced by vagal nerve stimulation in the guinea-pig.
    Fryer and Maclagan
    Eur.J.Pharmacol., 1987;139:187

Product Datasheets

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Citations for Ipratropium bromide

The citations listed below are publications that use Tocris products. Selected citations for Ipratropium bromide include:

3 Citations: Showing 1 - 3

  1. A subnanomolar concentration of Pituitary Adenylate Cyclase Activating Polypeptide (PACAP) pre-synaptically modulates glutamatergic transmission in the rat hippocampus acting through acetylcholine
    Authors: Valeria Et al.
    Neuroscience  2016;340:551
  2. Label-free cell phenotypic assessment of the biased agonism and efficacy of agonists at the endogenous muscarinic M3 receptors.
    Authors: Deng Et al.
    J Pharmacol Toxicol Methods  2013;68:323
  3. Neuregulin-1 enhances depolarization-induced GABA release.
    Authors: Woo Et al.
    Neuron  2007;54:599

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